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Together, our findings elucidate fundamental insight into the role of glutamine metabolism in not only regulating tumor growth and the TME but also controlling the generation and recruitment of MDSCs and reprogramming of TAMs, as well as regulating IDO expression, which results in inhibition of cancer growth, metastasis, and immune evasion

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Overexpression of hGSTA2-2 in K562 cells protects against hydrogen peroxide-induced apoptosis and inhibits JNK and caspase 3 activation

Large-scale deletion and point mutations of the nuclear NDUFV1 and NDUFS1 genes in mitochondrial complex I deficiency

Retinoids (Soriatine, acitretin): A form of vitamin A