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Some drugs can cause a glomerular lesion (e.g., pamidronate, non-steroidal anti-inflammatory drugs, anti-angiogenetic drugs), others preferentially target the tubule

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A 2014 review by Seiwerth and colleagues in Current Pharmaceutical Design summarized the preclinical vascular effects attributed to BPC-157 and framed the mechanistic hypotheses that subsequent studies have tested

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Of course, what both sides fail to understand is theyre tackling the wrong question entirely