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Shared epigenetic resistance mechanisms/targets of sorafenib and lenvatinib The epigenetic mechanisms underlying sorafenib/lenvatinib resistance encompass shared regulatory processes, including DNA/RNA methylation, histone modifications, and non-coding RNA regulation, and shared resistance outcomes involving dysregulated programmed cell death, metabolic reprogramming, enhanced cancer stem cell properties, and uncontrolled proliferative signaling pathways

Ward WK, LaCava EC, Paquette TL, Beard JC, Wallum BJ, Porte D (1987) Disproportionate elevation of immunoreactive proinsulin in type 2 (non-insulin-dependent) diabetes mellitus and in experimental insulin resistance

Anti-tumor activity of rosmarinic acid-loaded silk fibroin nanoparticles on HeLa and MCF-7 cells

[DOI] [PubMed] [Google Scholar] 34.Colazingari S., Treglia M., Najjar R., Bevilacqua A

24 h post-LPS challenge) dose-dependently enhances autophagy in LPS-challenged A549 cells, as shown by increased LC3-II and decreased p62 protein levels [6]

Additionally, several scientists showed that mitochondrial DNA is altered in aged and AD patients (Klein et al., 2021) due to the direct implication of A on mitochondria