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Recent evidence suggests that AST may interfere with these pathogenic processes by reducing amyloid- aggregation and attenuating tau hyperphosphorylation in Alzheimers models, thereby mitigating protein-induced neuronal toxicity

379 Such membrane perturbations can alter the sensitivity of the UPR machinery, linking folding stress not only to protein-specific defects but also to metabolic and lipid-associated conditions such as nonalcoholic fatty liver disease

doi: 10.59327/IPCC/AR6-9789291691647 65 JiangY

1 These frequent respiratory interruptions result in periods of low blood oxygen levels, nervous system dysfunction, and sleep fragmentation

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Testifying today is Board President Seung Oh, Board Member Renee Parker