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The premature arrest of brain growth in children with ASD has been observed [202], which can also be explained by abnormal activation of apoptosis
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In addition, TMAO was suggested to activate NFB, likely through a G-protein coupled receptor G signaling pathway in endothelial cells and vascular smooth muscle cells, leading to enhanced expression of proinflammatory cytokines and leukocyte adhesion to endothelial cells in vitro (Seldin et al., 2016)
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