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Proposes a structural mechanism by which APOA1 enhances cholesterol efflux capacity in smaller HDL particles compared to larger HDLs, involving the flipping of APOA1s C-terminal region to enable ABCA1 engagement
Recommendations for the Treatment of Dyslipidemia in Patients with Acute Coronary Syndrome (ACS) Table 30
In this pathophysiological context, de novo lipogenesis is thought to contribute to the origin of NAFLD, which is often associated with insulin resistance [78,170]
Vardi, Y., Gruenwald, I., Sprecher, E., Gertman, I
D.MullaneyB.SchillerB
J Biosci Bioeng 2014;118(4):47681
